Fat-Loss Peptides — Incretins, Amylin Analogues & Metabolics
The fat-loss shelf looks like one category but is really two. Most of the demand sits with the incretin receptor agonists — the GLP-1 class and its dual- and triple-agonist descendants — while a second group works through entirely different metabolic levers. Sorting the shelf by how a compound acts, rather than by headline weight-loss numbers, is the fastest way to land on the right vial. Everything here is the same Phiogen catalog, lot-tested and CoA-backed, so the decision is purely which mechanism you want.
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Retatrutide
Triple GLP-1/GIP/Glucagon receptor agonist
Tirzepatide
39-amino-acid synthetic peptide with C20 fatty diacid modification
Semaglutide
GLP-1 receptor agonist (31-amino acid lipidated peptide)
Cagrilintide
Long-acting amylin analog
Survodutide
GLP-1/Glucagon dual agonist (BI 456906)
Mazdutide
GLP-1/Glucagon dual receptor agonist
5-Amino-1MQ
5-Amino-1-methylquinolinium iodide
AOD9604
Tyr-hGH 177-191 fragment
BAM-15
Mitochondrial uncoupler small molecule
SLU-PP-332
ERR pan-agonist small molecule
Tesofensine
Triple monoamine reuptake inhibitor
Adipotide (FTPP)
FTPP proapoptotic peptidomimetic
Cagri-Reta
Cagri-Sema Blend
Long-acting amylin analog
L-Carnitine
(R)-3-Hydroxy-4-(trimethylammonio)butanoate
L-Carnitine 5-Pack
(R)-3-Hydroxy-4-(trimethylammonio)butanoate
Reta-Cagri
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How the fat-loss shelf splits: incretins vs everything else
The incretins are the compounds that reshaped this category — Semaglutide, Tirzepatide, Retatrutide, Mazdutide, Survodutide, Cagrilintide and the Cagri blends. They act on gut-hormone receptors to slow gastric emptying and blunt appetite centrally. The rest of the shelf — 5-Amino-1MQ, AOD9604, SLU-PP-332, BAM-15, Tesofensine, Adipotide, L-Carnitine — reaches fat loss through unrelated routes and shares none of the incretin GI-adaptation profile.
Choosing an incretin by receptor count
The single number that best predicts an incretin's effect is how many receptors it engages.
- One receptor (GLP-1): Semaglutide — the class benchmark, weekly dosing.
- Two (GLP-1 + GIP): Tirzepatide — GIP activity adds glucose handling and, in trials, eased the GI burden.
- Three (GLP-1 + GIP + glucagon): Retatrutide — glucagon adds energy expenditure on top of appetite suppression.
- Amylin, a separate pathway: Cagrilintide, and the Cagri-Sema / Cagri-Reta blends that pair it with an incretin backbone for two independent satiety mechanisms.
The non-incretin options
These are for buyers who want a different lever or can't use the GI-heavy incretins. 5-Amino-1MQ (oral, NNMT inhibition), SLU-PP-332 (an ERR agonist), BAM-15 (a mitochondrial uncoupler), AOD9604 (a GH-fragment), Tesofensine (a triple monoamine reuptake inhibitor) and L-Carnitine (fatty-acid transport) each work through their own mechanism, with far less human data behind them than the incretins.
Before your first vial
Match the compound to the mechanism you actually want, confirm the vial size against your escalation plan, and read the lot CoA. Incretin vial sizes are dosed in milligrams-per-week on a rising schedule, so a bigger vial is not automatically the better buy.
Fat Loss peptides — buying & shipping FAQ
What you need to know before ordering fat loss vials.
What actually separates Retatrutide from Tirzepatide?
Tirzepatide hits GLP-1 and GIP receptors; Retatrutide adds a third, glucagon. Glucagon activation raises energy expenditure independently of appetite, which is the mechanistic reason the triple-agonist trials posted larger figures than the dual-agonist ones. It is one more receptor, not a stronger version of the same drug.
Is Cagrilintide an incretin?
No — it is an amylin analogue. Amylin signals satiety through a hypothalamic pathway separate from GLP-1, which is why cagrilintide is combined with Semaglutide or Retatrutide (the Cagri-Sema and Cagri-Reta blends): two non-competing satiety mechanisms at once rather than a bigger dose of one.
Which fat-loss compounds are not incretins?
5-Amino-1MQ, SLU-PP-332, BAM-15, AOD9604, Tesofensine, Adipotide and L-Carnitine. Each works through its own mechanism — NNMT inhibition, ERR agonism, mitochondrial uncoupling, and so on — and none of them share the incretin GI-adaptation profile or its depth of human trial data.
Why do vial sizes not compare cleanly on this shelf?
Incretins are dosed in milligrams per week on an escalating schedule, and vials run from a few mg to 60 mg. A 5 mg vial of one compound and a 15 mg vial of another are not the same purchase, so compare on cost per milligram and on where each sits in your escalation, not on sticker price.
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